4 edition of Cells, membranes, and disease, including renal found in the catalog.
|Statement||edited by Eric Reid and G.M.W. Cook and J.P. Luzio.|
|Series||Methodological surveys in biochemistry and analysis ;, v. 17 (B), Methodological surveys in biochemistry and analysis ;, v. 17., Methodological surveys in biochemistry and analysis.|
|Contributions||Reid, Eric, 1922-, Cook, G. M. W. 1938-, Luzio, J. P.|
|LC Classifications||RB25 .I56 1986|
|The Physical Object|
|Pagination||xiv, 491 p. :|
|Number of Pages||491|
|LC Control Number||87022060|
Intrarenal regulatory T cells (Tregs), including those recruited to the kidney, have suppressive effects on both adaptive and innate immune cells, and probably also intrinsic kidney cells. Cell-extracellular matrix (ECM) interactions are essential for tissue development, homeostasis, and response to injury. Basement membranes (BMs) are specialized ECMs that separate epithelial or endothelial cells from stromal components and interact with cells via cellular receptors, including integrins and discoidin domain by: 9.
Apoptotic cell death is usually a response to the cell’s microenvironment. In the kidney, apoptosis contributes to parenchymal cell loss in the course of acute and chronic renal injury, but does not trigger an inflammatory response. What distinguishes necrosis from apoptosis is the rupture of the plasma membrane, so necrotic cell death is accompanied by the release of unprocessed Cited by: 1. Cell membranes are composed of hydrophobic lipids that repel water and aqueous solutes. The movement of solutes and water across cell membranes is made possible by discrete classes of integral membrane proteins, including channels, pumps, and transporters. Renal disease is associated with a reduction in functional nephrons. The rest of the.
The outer-membrane autotransporter proteins are also an effective mechanism of delivering virulence factors involved in colonization, disease progression, and immune system evasion. Other species associated with NGU include Mycoplasma genitalium, Ureaplasma urealyticum, and . Thin basement membrane disease (TBMD) is an inherited disorder that mainly affects the glomeruli, which are tiny tufts of capillaries (small blood vessels) in the kidneys that filter wastes from the blood. It is a rare disorder that has been diagnosed in less than 1 percent of the population.
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Cells, Membranes, and Disease, Including Renal It seems that you're in USA. We have a dedicated site for USA Membranes, membranes Disease, Including Renal Including Renal.
Editors: Reid, E., Effects of Ion Membranes on Cell Membranes and the Cytoskeleton. Pages Brand: Springer US. Complement-Mediated Plasma-Membrane Damage: Effects on Cell Physiology, Prospects of Cell Recovery and Implications for Disease Mechanisms J.
Paul Luzio, Arefaine Abraha, Peter J. Richardson, Richard A. Daw, Caroline A. Sewry, B. Paul Morgan et al. Get this from a library. Cells, membranes, and disease, including renal. cell recovery and implications for disease mechanisms.- #C-3 Erythrocyte membrane derangements associated with sickle cell disease.- #C-4 The membrane-cytoskeletal axis in phagocytosing PMN- leucocytes and virally transformed fibroblasts.- #NC(C) Supplementary Items.
Chronic kidney disease also is an integral part of the metabolic syndrome. Diabetes and hypertension are the two leading causes of renal failure, and kidney disease of any etiology leads to insulin resistance and its sequelae. Basement membranes (BMs) are specialized ECMs that separate epithelial or endothelial cells from stromal components and interact with cells via cellular receptors, including integrins and discoidin domain receptors.
Disruption of cell–BM interactions due to either injury or genetic defects in either the ECM components or cellular receptors often lead to irreversible tissue injury and loss of organ by: 9. Discusses significant advances in the field—including those related to pathophysiology of glomerular diseases, electrolyte disorders, renal tubular transport systems, hypertension, transplantation, hereditary diseases, and chronic kidney disease—to keep your knowledge current.
The glomerular basement membrane (GBM) mostly contains α, which is also present in the eye, ear and lungs. α is found in skin basement membrane and parts of the gut. Tissue-specific collagen and laminin isoforms replace generic forms during kidney : Eleri W.
Williams, A. Neil Turner. Practice cell membranes and trafficking disorders with Khan Academy's free online exercises.
If you're seeing this message, it means we're having trouble loading external resources on our website. If you're behind a web filter, please make sure that the domains * and * are unblocked. Clin Physiol Biochem. ;4(5) Role of membranes in disease.
Goldberg DM, Riordan JR. The membranes of mammalian cells are composed of an ordered array of lipids and proteins, the latter containing carbohydrate residues directed towards the exterior and important in the interaction of cells with each other and with external by: 6.
Although the major focus of this article is on urinary exosomes, it is likely that exosomes secreted into the blood and extracellular fluid have roles in renal physiology and pathophysiology, especially among cell types with their plasma membranes in direct contact with the vascular compartment such as cells of the immune system and endothelial by: Polycystic kidney disease is a common genetic disorder in which fluid-filled cysts displace normal renal tubules.
Here we focus on autosomal dominant polycystic kidney disease, which is attributable to mutations in the PKD1 and PKD2 genes and which is characterized by perturbations of renal epithelial cell growth control, fluid transport, and by: Comprehensive and up to date, the third edition of Diagnostic Pathology: Kidney Diseases, written by Robert B.
Colvin, MD and Anthony Chang, MD, expertly covers all aspects of common and rare renal diseases and their variants. This easy-to-use, point-of-care reference offers a state-of-the-art, concise presentation of major pathological, clinical, pathophysiological, and genetic Format: Book.
Aquaporins (AQPs) are a family of highly selective transmembrane channels that mainly transport water across the cell and some facilitate low-molecular-weight solutes.
Eight AQPs, including AQP1, AQP2, AQP3, AQP4, AQP5, AQP6, AQP7, and AQP11, are expressed in different segments and various cells in the kidney to maintain normal urine concentration function. AQP2 is critical in regulating urine Cited by: 3.
Cilia are microtubule-based organelles, protruding from the apical cell surface and anchoring to the cytoskeleton. Primary (nonmotile) cilia of the kidney act as mechanosensors of nephron cells, responding to fluid movements by triggering signal transduction.
The impaired functioning of primary cilia leads to formation of cysts which in turn contribute to development of diverse renal diseases. The prevalence of renal diseases is emerging as a public health problem.
Despite major progress in supportive therapy, mortality rates among patients remain high. In an attempt to find innovative treatments to stimulate kidney regeneration, stem cell-based technology has been proposed as a potentially promising strategy.
Here, we summarise the renoprotective potential of pluripotent and Cited by: 6. Introduction. The kidneys have several essential homeostatic functions. These functions include waste removal (NH3), fluid/electrolyte balance, metabolic blood acid-base balance, as well as producing/modifying hormones for blood pressure, calcium/potassium homeostasis, and red blood cell production.
The renal corpuscle (filtration unit, which comprises the glomerulus and the surrounding glomerular or Bowman’s capsule) and tubules (reabsorption and excretion.
According to Yao’s observation, CX3CR1 was expressed in human clear cell Renal Cell Carcinoma (RCC) cell lines, and only membrane positive cells were responsible for CX3CL1‐induced cell migration. Extracellular signal-Related Kinases (ERK1/2) and PI3K/Akt were triggered by soluble CX3CL1 dependent on by: 4.
Methods in Kidney Cell Biology, Part B, Volume represents state-of-the-art techniques in renal research that are ideal for veterans, graduate students, postdoctoral fellows, clinical scientists and principal investigators.
Topics in the new release include Single glomerular proteomics – a novel method in translational glomerular cell biology, Measurement of cytosolic and intraciliary.
Cell membrane diseases are life-threatening disorders that are genetic in nature, and they usually work against proteins in our body that are key to ion channels and various receptors within the membrane. These diseases work by either disrupting the normal functions of the cells or by simply affecting the cell membrane.
Structures in the kidney include the renal corpuscle (comprised of a glomerulus and bowman's capsule), which filters the blood, and various tubules, through which blood and urine pass. The renal corpuscle is made of parietal cells, podocytes and mesangial cells.
The various tubules are made of columnar and cuboidal epithelial cells. Mesangial cells. Renal cell carcinoma (RCC) is a kidney cancer that originates in the lining of the proximal convoluted tubule, a part of the very small tubes in the kidney that transport primary is the most common type of kidney cancer in adults, responsible for approximately 90–95% of cases.
Initial treatment is most commonly either partial or complete removal of the affected kidney(s).Specialty: Oncology.This review explores the current evidence that ILA and Th17 cells may be pathogenic in immune kidney disease, including anti-glomerular basement membrane disease, antineutrophil cytoplasmic antibody associated vasculitis and lupus nephritis, as well as in acute kidney injury.
3. Role of MCs in glomerular pathology. Glomerular diseases manifest as diverse clinical syndromes and etiologies are not clearly understood. Glomerular changes are complex, involving all glomerular cell types including MCs, endothelial cells, podocytes, parietal epithelial cells and infiltrating inflammatory cells [2, 6–9].Deposition of immunoglobulins (Ig) or immune complexes in the Cited by: